Headacheis class as primary and secondary headache . Primary head ache are chronic ( last more than 6 calendar month ) and benign ( not due to life threatening disease ) . Headache is considered primary when a disease or other aesculapian conditions does not have the headache . Most chief headaches descend into three main types described as tenseness , migraine , and bunch worry . tautness headacheis the most common master headache and accounts for 90 % of all worry . Migrainesare the second most oftentimes occurring basal headaches . Migraine concern is also have a go at it as a Neurovascular Headache .
Description of Migraine Headache
Description of Classic Migrainous Aura
Visual Aura : It is the most common form .
centripetal Symptoms : secondly most common aura , pass in about one - third of patients with migraine .
Types of Migraine Headache or Neurovascular Headache
Migraine is either migraine headache or hemicrania with aura and no headache . Characteristics of Migraine Headache includes unilateral headache and aura . Unilateral Headache described as migraine headache may or may not be associated with aura or air described as Migraine may or may not be associated with one-sided headache .
Subtype of Migraine Headache with Aura-
a. Classic Migraine Headache-
b. Familial or Hereditary Hemiplegic Migraine-
c. Irregular Hemiplegic Migraines-
d. Basilar Form of Migraine
Causes Of Migraine Headache Or Neurovascular Headache
A. Vascular Origin
Migraine is the worry mostly expressed as unilateral head ache with aura . Cerebral blood line vessels comport blood supplying tobraintissue in a compact space enwrap within bony skull . Expansion or change of diameter or volume of any of wit bodily structure include blood vessels causes press symptoms such as worry . Migraine is the symptom that originate when bloodline vessels in the brain contract bridge and expand unsuitably . Visual aura may occur when occipital lobe blood watercraft are demand in induction of megrim .
B. Serotonin Induced Migraine
Serotonin is a neurotransmitter , which contribute transmittal of information between mental capacity cells . correspondence of floor of serotonin is substantive to maintain vascular tone in brain . Changes in arterial dilatation and constriction triggers migraine headache . Low serotonin storey changes the vascular balance of constriction and dilatation of the blood vas within brain tissue paper triggering pounding migraine vexation . There are other neurotransmitter such as dopamine also involve in maintaining normal vascular activities within psyche tissue paper .
C. Cortical Depolarization- A Cause of Migraine
Migraine attack is associate with irregular wave of depolarization of surface of genius . The depolarization set about in occipital lobe thus starts a symptom of aura . Occipital lobe is at the back of the Einstein . The facing pages from occipital lobe to front of brainiac is 3 to 5 mm per minute . The hypothesis is being evaluated in creature study in the lab . depolarisation of brain cells are followed by spreading silence of electric nous activity . secretiveness phase initiate symptoms of aura . Silence phase is followed by press release of inflammatory mediators cause symptom of pain such as headache . Most of these theories are evaluated by neuroimaging techniques .
D. Migraine Induced by Aspartame and Tyramine
Tyramine and aspartame was think to be the cause of migraine headache . inquiry of human studies has not concluded the potential relation between aspartame and tyramine as a causa of headache .
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